Statins- decreasing achilles tendon thickness

Statins used without the use of the nutrient Coenzyme Q10 should be against the law. Maybe statins should be taken back to the drawing board and not used at all.

CoQ10 is used for energy production of cells. So when the cell doesn't have enough energy it will not create the important building blocks it should. Statins stop production of CoQ10 a vital part of the energy production process. Therefore cells will not build collagen for example. Collagen is a protein structure that is the connective tissue of our body. If the connective tissue is not healthy then injury could easily occur. If you have a thinner tendon in your achilles may not be strong enough to hold your weight and you may be injured, just by walking and it may cause more

Take a more natural route to decrease the cholesterol.

Regression of Achilles tendon thickness after statin treatment in patients with familial hypercholesterolemia: An ultrasonographic study; Tsouli SG, Xydis V, Argyropoulou MI, Tselepis AD, Elisaf M, Kiortsis DN; Atherosclerosis (Nov 2008)

OBJECTIVE: Achilles tendon xanthomas (ATX) have been associated with increased cardiovascular risk in patients with familial hypercholesterolemia (FH). The aim of this study was to evaluate clinical and ultrasonographic changes of ATX in patients with FH under statin treatment. METHODS: Achilles tendon thickness (ATT) and echostructure were studied by ultrasonography (US) in 80 unrelated heterozygous FH patients and in 80 age- and sex-matched controls. For ATT measurements the anterioposterior diameter (mm) of the Achilles tendon was measured on sagittal scans. Patients were treated with atorvastatin (mean dose 20+/-10mg/day) and a follow-up examination was performed 12 months later. RESULTS: Clinical examination revealed xanthomas in 15 patients. On US normal fibrillar echostructure (grade 1) of the Achilles tendon (AT) was observed in 42 patients, abnormal echostructure with diffuse heterogeneous echo pattern (grade 2) in 30 patients and focal hypoechoic lesions (grade 3) in 8 patients. At baseline, ATT of all patients (5.23+/-0.91mm) was significantly larger compared with controls (4.20+/-0.70mm) (p<0.05). Patients with grades 2 (5.20+/-0.60mm) and 3 (6.98+/-1.07mm) had significantly larger ATT than those with grade 1 (4.90+/-0.55mm), p<0.05. Patients with grade 1 showed significant reduction in ATT after statin treatment (from 4.90+/-0.55mm to 4.50+/-0.43mm, p<0.01). In patients with grades 2 and 3 abnormal echostructure remained unchanged and no significant reduction in ATT was observed. CONCLUSION: Statin treatment reduces ATT in FH patients with normal AT echostructure. Ultrasound detects AT structural involvement and is useful in the monitoring of response to treatment.